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Cited by 122 publications
(59 citation statements)
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“…26,27 Gynoidal fat distribution has been associated with a lower risk of cardiovascular disease than when the fat deposition distribution is androidal. 48 It appears that the association of fat deposition with cardiovascular disease risk factors is not directly related to overall obesity; rather, the distribution of the fat depositions is important.…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…26,27 Gynoidal fat distribution has been associated with a lower risk of cardiovascular disease than when the fat deposition distribution is androidal. 48 It appears that the association of fat deposition with cardiovascular disease risk factors is not directly related to overall obesity; rather, the distribution of the fat depositions is important.…”
Section: Discussionmentioning
confidence: 99%
“…[22][23][24][25] Several DD2R gene polymorphisms, including the TaqI polymorphism located Ϸ10 kb away from the gene, 25 have been associated with several psychiatric disorders related to stimulation of the reward pathway, including substance abuse. 23,24 Polymorphisms in the receptor gene have also been associ-ated with obesity-related parameters such as body weight 26 and adult-onset obesity and dietary carbohydrate preference. 27 In populations with a previously low prevalence of cardiovascular risk factors, such as in Hong Kong, the effects of genetic factors may be particularly evident in the determination of which individuals develop disorders such as obesity, hypertension, and type 2 diabetes mellitus.…”
mentioning
confidence: 99%
“…[6][7][8][9][10] Furthermore, the dopaminergic system seems to play a role in the development of obesity, presumably via regulation of food intake. 6,[11][12][13] We have recently reported that the…”
Section: Journal Of Human Hypertension (2001) 15 553-558mentioning
confidence: 99%
“…The Cys 311 variant of the human DRD2 has been shown to markedly impair the ability of the receptor to modulate adenylyl cyclase activity (6). Studies in genetically heterogeneous human populations have shown weak associations between DRD2 variants and obesity (7)(8)(9)(10)(11).We have previously observed that the location of DRD2 on chromosome 11q23-24 is near the peak of linkage of a locus known to influence type 2 diabetes, obesity, and energy expenditure in Pima Indians (12,13) and that individuals with a Cys311-encoding allele have a higher BMI than those homozygous for the Ser311-encoding allele (14). Therefore, the aim of the present study was to assess whether the Cys 311 variant of DRD2, a plausible candidate gene for obesity, promotes body weight gain by inhibiting energy expenditure in humans.…”
mentioning
confidence: 99%